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Three more cases of CWD found in free-ranging deer in Macon County

Posted Mar 25 2012 11:12am
Three more cases of CWD found in free-ranging deer in Macon County

Statewide

Published on: Mar. 23, 2012

Posted by Joe Jerek

JEFFERSON CITY Mo – The Missouri Department of Conservation (MDC) received final test results today from tissue samples taken from 656 free-ranging deer harvested earlier this year. Results included three CWD-positives with two from adult does and one from an adult buck. The testing was conducted specifically for CWD sampling by MDC staff and area landowners over a 163-square-mile area in northeast Linn and northwest Macon counties.

Missouri’s first two cases of CWD in free-ranging deer were detected in two adult bucks harvested in northwest Macon County during the 2011 fall firearms deer season. The three most recent CWD positives were harvested within two miles of the two original cases of CWD.

MDC conducted its 2011 fall tissue-sampling effort in response to two cases of CWD found in captive white-tailed deer at two private, captive-hunting preserves in Macon and Linn counties. Since October 2011, three more captive deer at the Macon County preserve have tested positive for CWD. Depopulation, quarantine and other management activities at the private preserve are being coordinated by the Missouri Department of Agriculture. The five cases of CWD in free-ranging deer have been found within two miles of the Macon County preserve.

While MDC identified Missouri's first two cases of CWD in free-ranging deer from test results received in January, the Department has been testing for it for years. With the help of hunters, MDC has tested more than 34,000 free-ranging deer for CWD from all parts of the state since 2002.

MDC staff are further analyzing recent test results, continuing to evaluate efforts and lessons learned from other states with CWD, and consulting with various other wildlife experts around the country. The Department’s main objectives are to limit the prevalence and restrict the spread of CWD in Missouri.

According to MDC Deer Biologist Jason Sumners, there are several main factors associated with the management of free-ranging deer that will influence the future prevalence and distribution of CWD in Missouri: local deer density, deer concentration, the movement of deer and the movement of deer carcasses.

“Yearling and adult male deer have been found to exhibit CWD at a much higher rate than yearling and adult females,” Sumners said. “Of the 10 cases of CWD identified in both captive and free-ranging deer in Missouri, eight have been in adult bucks. Additionally, dispersal of yearling males from the ranges where they were born is one of the most likely means of expanding the distribution of CWD. The movement of infectious materials in the form of hunter-harvested deer carcasses that contain heads and spinal columns, where the disease concentrates, may also serve as a means of introducing CWD to other regions of the state.”

Sumners added that CWD has been found in only one small pocket of the state. “Our management efforts will focus on minimizing the prevalence and preventing the further spread of the disease from the area. We will keep the public informed as we develop those efforts.”

CWD is a neurological disease that is limited to deer, elk, moose and other members of the deer family, known as “cervids.” CWD can only be confirmed in deer by laboratory testing of the brain stem or lymph tissue. CWD is transmitted through prions, which are abnormal proteins that attack the nervous systems of these species. These prions accumulate in the brain, spinal cord, eyes, spleen, and lymph nodes of infected animals. CWD is spread by animal-to-animal contact or by animal contact with soil that contains prions from urine, feces or the decomposition of an infected animal.

Deer and other cervids with signs of CWD show changes in natural behavior and can exhibit extreme weight loss, excessive salivation, stumbling and tremors. CWD can be present in and transmitted by infected animals for up to several years before symptoms of the disease appear.

“Most deer that test positive for CWD appear to be healthy,” Sumners said. “Although one of the free-ranging does that tested positive for CWD did exhibit clinical signs of the disease.”

The Missouri Department of Agriculture (MDA) says there is no evidence from existing research that CWD can spread to domestic livestock, such as sheep or cattle. The Missouri Department of Health and Senior Services (MDHSS) says there is no scientific evidence that CWD is transmissible to humans through contact with or the consumption of deer meat.

The disease was first recognized in 1967 in captive mule deer in Colorado. CWD has been documented in both captive and free-ranging deer in Missouri, along with neighboring Kansas and Nebraska. It has been documented in free-ranging deer in neighboring Illinois, and in captive elk in neighboring Oklahoma. CWD has also been documented in both captive and free-ranging members of the deer-family in Colorado, Minnesota, New York, South Dakota, Wisconsin and the Canadian provinces of Alberta and Saskatchewan. Maryland, New Mexico, North Dakota, Utah, Virginia, West Virginia and Wyoming also have documented cases of CWD in free-ranging members of the deer family. Michigan and Montana have documented cases of CWD in captive members of the deer family.



http://mdc.mo.gov/newsroom/three-more-cases-cwd-found-free-ranging-deer-macon-county





Tuesday, January 24, 2012

CWD found in two free-ranging deer from Macon County Missouri



http://chronic-wasting-disease.blogspot.com/2012/01/cwd-found-in-two-free-ranging-deer-from.html




Friday, October 21, 2011



Chronic Wasting Disease Found in Captive Deer Missouri



http://chronic-wasting-disease.blogspot.com/2011/10/chronic-wasting-disease-found-in.html




Friday, February 26, 2010



Chronic wasting disease found in Missouri deer



http://chronic-wasting-disease.blogspot.com/2010/02/chronic-wasting-disease-found-in.html




WHICH CAME FIRST, THE CART OR THE HORSE ??



Minnesota

CAPTIVE CWD CONFIRMED 2002



FREE RANGING CWD CONFIRMED 2011



http://wwwnc.cdc.gov/eid/article/18/3/11-0685-f1.htm





Colorado

Captive CWD discovered 1967

Free ranging CWD discovered 1981



PLEASE STUDY THIS MAP !



SEE CWD MAP, RELATE TO DATES OF GAME FARM INFECTION, TO DATE OF INFECTION RATE IN WILD, SURROUNDING SAID INFECTED GAME FARMS. ...TSS


http://wwwnc.cdc.gov/eid/article/18/3/11-0685-f1.htm





*** Chronic Wasting Disease CWD CDC REPORT MARCH 2012 ***



Saturday, February 18, 2012

Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

CDC Volume 18, Number 3—March 2012

SNIP...

Long-term effects of CWD on cervid populations and ecosystems remain unclear as the disease continues to spread and prevalence increases. In captive herds, CWD might persist at high levels and lead to complete herd destruction in the absence of human culling. Epidemiologic modeling suggests the disease could have severe effects on free-ranging deer populations, depending on hunting policies and environmental persistence (8,9). CWD has been associated with large decreases in free-ranging mule deer populations in an area of high CWD prevalence (Boulder, Colorado, USA) (5).

SNIP...

Reasons for Caution There are several reasons for caution with respect to zoonotic and interspecies CWD transmission. First, there is strong evidence that distinct CWD strains exist (36). Prion strains are distinguished by varied incubation periods, clinical symptoms, PrPSc conformations, and CNS PrPSc depositions (3,32). Strains have been identified in other natural prion diseases, including scrapie, BSE, and CJD (3). Intraspecies and interspecies transmission of prions from CWD-positive deer and elk isolates resulted in identification of >2 strains of CWD in rodent models (36), indicating that CWD strains likely exist in cervids. However, nothing is currently known about natural distribution and prevalence of CWD strains. Currently, host range and pathogenicity vary with prion strain (28,37). Therefore, zoonotic potential of CWD may also vary with CWD strain. In addition, diversity in host (cervid) and target (e.g., human) genotypes further complicates definitive findings of zoonotic and interspecies transmission potentials of CWD. Intraspecies and interspecies passage of the CWD agent may also increase the risk for zoonotic CWD transmission. The CWD prion agent is undergoing serial passage naturally as the disease continues to emerge. In vitro and in vivo intraspecies transmission of the CWD agent yields PrPSc with an increased capacity to convert human PrPc to PrPSc (30). Interspecies prion transmission can alter CWD host range (38) and yield multiple novel prion strains (3,28). The potential for interspecies CWD transmission (by cohabitating mammals) will only increase as the disease spreads and CWD prions continue to be shed into the environment. This environmental passage itself may alter CWD prions or exert selective pressures on CWD strain mixtures by interactions with soil, which are known to vary with prion strain (25), or exposure to environmental or gut degradation. Given that prion disease in humans can be difficult to diagnose and the asymptomatic incubation period can last decades, continued research, epidemiologic surveillance, and caution in handling risky material remain prudent as CWD continues to spread and the opportunity for interspecies transmission increases. Otherwise, similar to what occurred in the United Kingdom after detection of variant CJD and its subsequent link to BSE, years of prevention could be lost if zoonotic transmission of CWD is subsequently identified, SNIP...SEE FULL TEXT ;



*** Chronic Wasting Disease CWD CDC REPORT MARCH 2012 ***

Saturday, February 18, 2012

Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

CDC Volume 18, Number 3—March 2012

http://wwwnc.cdc.gov/eid/article/18/3/11-0685_article.htm



see much more here ;




http://chronic-wasting-disease.blogspot.com/2012/02/occurrence-transmission-and-zoonotic.html






Sunday, January 22, 2012

Chronic Wasting Disease CWD cervids interspecies transmission

http://chronic-wasting-disease.blogspot.com/2012/01/chronic-wasting-disease-cwd-cervids.html





Thursday, January 26, 2012

The Risk of Prion Zoonoses

Science 27 January 2012: Vol. 335 no. 6067 pp. 411-413 DOI: 10.1126/science.1218167

http://transmissiblespongiformencephalopathy.blogspot.com/2012/01/risk-of-prion-zoonoses.html





Thursday, January 26, 2012

Facilitated Cross-Species Transmission of Prions in Extraneural Tissue

Science 27 January 2012: Vol. 335 no. 6067 pp. 472-475 DOI: 10.1126/science.1215659

http://transmissiblespongiformencephalopathy.blogspot.com/2012/01/facilitated-cross-species-transmission.html





CJD9/10022

October 1994

Mr R.N. Elmhirst Chairman British Deer Farmers Association Holly Lodge Spencers Lane BerksWell Coventry CV7 7BZ

Dear Mr Elmhirst,

CREUTZFELDT-JAKOB DISEASE (CJD) SURVEILLANCE UNIT REPORT

Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.

The Surveillance Unit is a completely independant outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.

The Chief Medical Officer has undertaken to keep the public fully informed of the results of any research in respect of CJD. This report was entirely the work of the unit and was produced completely independantly of the the Department.

The statistical results reqarding the consumption of venison was put into perspective in the body of the report and was not mentioned at all in the press release. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of venison was highlighted only once by the media ie. in the News at one television proqramme.

I believe that a further statement about the report, or indeed statistical links between CJD and consumption of venison, would increase, and quite possibly give damaging credence, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all.


http://web.archive.org/web/20030511010117/http://www.bseinquiry.gov.uk/files/yb/1994/10/00003001.pdf





THIRD CJD REPORT UK 1994

snip...

Consumption of venison and veal was much less widespread among both cases and controls. For both of these meats, there was evidence of a trend with increasing frequency of consumption being associated with increasing risk of CJD. These associations were largely unchanged when attention was restricted to pairs with data obtained from relatives. ...


http://www.cjd.ed.ac.uk/Archive%20reports/report3.pdf




Saturday, March 10, 2012

Enhanced Surveillance Strategies for Detecting and Monitoring Chronic Wasting Disease in Free-Ranging Cervids Open-File Report 2012–1036 National Wildlife Health Center

Open-File Report 2012–1036

http://chronic-wasting-disease.blogspot.com/2012/03/enhanced-surveillance-strategies-for.html




a few things to consider please. one, CWD has already been transmitted to many cattle in the lab (86% in one study). the oral route would have a much longer incubation period, but we already know that CWD will transmit back to cervids via the oral route, very efficiently. the threat of spreading CWD via close contact, like at feeding grounds is great. every bit of science to date shows this. so to congregate deer together by unnatural means is not smart in my opinion. another fear has come to pass as well, another strain of CWD, yes a second strain. and just recently science has shown that a natural case of BSE has been transmitted to a GOAT. These findings demonstrate that when CWD is directly inoculated into the brain of cattle, 86% of inoculated cattle develop clinical signs of the disease.

http://www.ars.usda.gov/research/publications/publications.htm?seq_no_115=194089





Thursday, February 09, 2012

50 GAME FARMS IN USA INFECTED WITH CHRONIC WASTING DISEASE

http://chronic-wasting-disease.blogspot.com/2012/02/50-game-farms-to-date-in-usa-infected.html






and when these game farms claim they are testing, and everything is o.k., think again...




Saturday, February 04, 2012

Wisconsin 16 age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised

http://chronic-wasting-disease.blogspot.com/2012/02/wisconsin-16-age-limit-on-testing-dead.html





National Wildlife Health Center

Open-File Report 2012–1036

U.S.

Enhanced Surveillance Strategies for Detecting and Monitoring Chronic Wasting Disease in Free-Ranging Cervids

snip...

In addition to locations of known CWD-positive individuals, other spatial risk factors related to CWD exposure should be considered. For example, the risk of free-ranging animals being exposed to CWD is likely greater in areas where captive cervid facilities have or had CWD-positive animals. Current evidence indicates that CWD infection rates are much higher in captive facilities than in wild populations (Keane and others, 2008), and perhaps this is driven by environmental contamination (Miller and others, 2006). This higher rate of infection in captive animals can increase the risk of disease exposure to surrounding wild populations. Furthermore, movement of infectious animals, carcasses, or other materials across the landscape, naturally or with human assistance, likely increases the risk to uninfected populations. The frequent movement of farmed elk (Cervus elaphus) and deer between production facilities, the concentration of infected animals on some facilities, and the possibility of their escape into the wild increases the risk of spreading CWD to uninfected populations of free-ranging animals. Because the infectious prions may persist in the environment for long periods, the introduction of either captive or free-ranging uninfected animals into a contaminated environment could increase their risk of infection. For example, locations from which sheep have been removed may remain contaminated with scrapie agent for more than 15 years (Georgsson and others, 2006).



Saturday, March 10, 2012

Enhanced Surveillance Strategies for Detecting and Monitoring Chronic Wasting Disease in Free-Ranging Cervids Open-File Report 2012–1036 National Wildlife Health Center

Open-File Report 2012–1036

http://chronic-wasting-disease.blogspot.com/2012/03/enhanced-surveillance-strategies-for.html




UPDATED CORRESPONDENCE FROM AUTHORS OF THIS STUDY I.E. COLBY, PRUSINER ET AL, ABOUT MY CONCERNS OF THE DISCREPANCY BETWEEN THEIR FIGURES AND MY FIGURES OF THE STUDIES ON CWD TRANSMISSION TO CATTLE ;

----- Original Message -----

From: David Colby

To: flounder9@verizon.net

Cc: stanley@XXXXXXXX

Sent: Tuesday, March 01, 2011 8:25 AM

Subject: Re: FW: re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 + Author Affiliations

Dear Terry Singeltary,

Thank you for your correspondence regarding the review article Stanley Prusiner and I recently wrote for Cold Spring Harbor Perspectives. Dr. Prusiner asked that I reply to your message due to his busy schedule. We agree that the transmission of CWD prions to beef livestock would be a troubling development and assessing that risk is important. In our article, we cite a peer-reviewed publication reporting confirmed cases of laboratory transmission based on stringent criteria. The less stringent criteria for transmission described in the abstract you refer to lead to the discrepancy between your numbers and ours and thus the interpretation of the transmission rate. We stand by our assessment of the literature--namely that the transmission rate of CWD to bovines appears relatively low, but we recognize that even a low transmission rate could have important implications for public health and we thank you for bringing attention to this matter.

Warm Regards, David Colby

--

David Colby, PhDAssistant ProfessorDepartment of Chemical EngineeringUniversity of Delaware



====================END...TSS==============


SNIP...SEE FULL TEXT ;

http://betaamyloidcjd.blogspot.com/2011/01/enlarging-spectrum-of-prion-like.html





UPDATED DATA ON 2ND CWD STRAIN

Wednesday, September 08, 2010

CWD PRION CONGRESS SEPTEMBER 8-11 2010

http://chronic-wasting-disease.blogspot.com/2010/09/cwd-prion-2010.html



Wednesday, November 16, 2011

Wisconsin Creutzfeldt Jakob Disease, CWD, TSE, PRION REPORTING 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/11/wisconsin-creutzfeldt-jakob-disease-cwd.html



Sunday, November 13, 2011

COLORADO CWD CJD TSE PRION REPORTING 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/11/colorado-cwd-cjd-tse-prion-reporting.html



PLUS, THE CDC DID NOT PUT THIS WARNING OUT FOR THE WELL BEING OF THE DEER AND ELK ;



Thursday, May 26, 2011

Travel History, Hunting, and Venison Consumption Related to Prion Disease Exposure, 2006-2007 FoodNet Population Survey

Journal of the American Dietetic Association Volume 111, Issue 6 , Pages 858-863, June 2011.

http://transmissiblespongiformencephalopathy.blogspot.com/2011/05/travel-history-hunting-and-venison.html



NOR IS THE FDA recalling this CWD positive elk meat for the well being of the dead elk ;


Wednesday, March 18, 2009

Noah's Ark Holding, LLC, Dawson, MN RECALL Elk products contain meat derived from an elk confirmed to have CWD NV, CA, TX, CO, NY, UT, FL, OK RECALLS AND FIELD CORRECTIONS: FOODS CLASS II

http://chronic-wasting-disease.blogspot.com/2009/03/noahs-ark-holding-llc-dawson-mn-recall.html




now, let’s see what the authors said about this casual link, personal communications years ago. see where it is stated NO STRONG evidence. so, does this mean there IS casual evidence ??




“Our conclusion stating that we found no strong evidence of CWD transmission to humans”



From: TSS (216-119-163-189.ipset45.wt.net)

Subject: CWD aka MAD DEER/ELK TO HUMANS ??

Date: September 30, 2002 at 7:06 am PST

From: "Belay, Ermias"

To:

Cc: "Race, Richard (NIH)" ; ; "Belay, Ermias"

Sent: Monday, September 30, 2002 9:22 AM

Subject: RE: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS

Dear Sir/Madam,

In the Archives of Neurology you quoted (the abstract of which was attached to your email), we did not say CWD in humans will present like variant CJD.

That assumption would be wrong. I encourage you to read the whole article and call me if you have questions or need more clarification (phone: 404-639-3091). Also, we do not claim that "no-one has ever been infected with prion disease from eating venison." Our conclusion stating that we found no strong evidence of CWD transmission to humans in the article you quoted or in any other forum is limited to the patients we investigated.

Ermias Belay, M.D. Centers for Disease Control and Prevention

-----Original Message-----

From:

Sent: Sunday, September 29, 2002 10:15 AM

To: rr26k@nih.gov; rrace@niaid.nih.gov; ebb8@CDC.GOV

Subject: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS

Sunday, November 10, 2002 6:26 PM ......snip........end..............TSS

Thursday, April 03, 2008

A prion disease of cervids: Chronic wasting disease

2008 1: Vet Res. 2008 Apr 3;39(4):41

A prion disease of cervids: Chronic wasting disease

Sigurdson CJ.

snip...

*** twenty-seven CJD patients who regularly consumed venison were reported to the Surveillance Center***,

snip...

full text ;


http://chronic-wasting-disease.blogspot.com/2008/04/prion-disease-of-cervids-chronic.html




CWD ongoing experiment on humans, long term $$$



Monday, November 14, 2011

WYOMING Creutzfeldt Jakob Disease, CWD, TSE, PRION REPORTING 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/11/wyoming-creutzfeldt-jakob-disease-cwd.html




Wednesday, November 16, 2011

Wisconsin Creutzfeldt Jakob Disease, CWD, TSE, PRION REPORTING 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/11/wisconsin-creutzfeldt-jakob-disease-cwd.html





Sunday, November 13, 2011

COLORADO CWD CJD TSE PRION REPORTING 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/11/colorado-cwd-cjd-tse-prion-reporting.html







Sunday, February 12, 2012

National Prion Disease Pathology Surveillance Center Cases Examined1 (August 19, 2011) including Texas

http://transmissiblespongiformencephalopathy.blogspot.com/2012/02/national-prion-disease-pathology.html




Saturday, March 5, 2011

MAD COW ATYPICAL CJD PRION TSE CASES WITH CLASSIFICATIONS PENDING ON THE RISE IN NORTH AMERICA

http://transmissiblespongiformencephalopathy.blogspot.com/2011/03/mad-cow-atypical-cjd-prion-tse-cases.html





PLEASE REMEMBER ;

The Akron, Ohio-based CJD Foundation said the Center for Disease Control revised that number in October of 2004 to about one in 9,000 CJD cases per year in the population group age 55 and older.

HAVE YOU GOT YOUR CJD QUESTIONNAIRE ASKING REAL QUESTIONS PERTAINING TO ROUTE AND SOURCE OF THE TSE AGENT THAT KILLED YOUR LOVED ONE ??

if not, why not...



Friday, November 30, 2007

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION

http://cjdquestionnaire.blogspot.com/2007/11/cjd-questionnaire.html




http://cjdquestionnaire.blogspot.com/





Terry S. Singeltary Sr. on the Creutzfeldt-Jakob Disease Public Health Crisis



http://www.youtube.com/watch?v=zf3lfz9NrT4


http://www.youtube.com/watch?v=c0tWkNvhO4g


http://www.youtube.com/watch?v=zf3lfz9NrT4&feature=results_main&playnext=1&list=PL780BE2AF0B62A944


full text with source references ;


http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/terry-singeltary-sr-on-creutzfeldt.html



TSS
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