Sent: Thursday, January 21, 2010 3:31 PM Subject: Kansas has more CWD cases
Kansas has more CWD cases
Four more deer killed in northwest Kansas are suspected of having chronic wasting disease. That’s in addition to six deer reported to have the disease about three weeks ago.
Mike Miller, Kansas Department of Wildlife and Parks information chief, said the deer were shot during recent deer seasons. Tissue samples were taken from about 2,300 deer for testing at a K-State laboratory. Most came from the 2009 deer seasons. Slightly more than half have been tested.
The four recently reported as positive at K-State have been sent to Iowa for federal verification. The six sent to Iowa earlier this month were confirmed to have the disease.
Chronic wasting disease is always fatal in deer, elk and moose. It hasn’t been documented in livestock, pets or humans.
It was first identified along the Colorado/Wyoming border about 40 years ago. Kansas’ first case was in 2005 in the extreme northwest corner of the state. Ten deer tested positive for CWD last year.
It appears to be moving eastward and southward.
One of the four that recently tested positive at K-State was the first-ever from Logan County. By Michael Pearce
Susceptibilities of Nonhuman Primates to Chronic Wasting Disease
From: TSS (216-119-163-189.ipset45.wt.net)
Subject: CWD aka MAD DEER/ELK TO HUMANS ???
Date: September 30, 2002 at 7:06 am PST
From: “Belay, Ermias”
Cc: “Race, Richard (NIH)” ; ; “Belay,
Sent: Monday, September 30, 2002 9:22 AM
Subject: RE: TO CDC AND NIH – PUB MED- 3 MORE DEATHS – CWD – YOUNG HUNTERS
In the Archives of Neurology you quoted (the abstract of which was attached to your email), we did not say CWD in humans will present like variant CJD.
That assumption would be wrong. I encourage you to read the whole article and call me if you have questions or need more clarification (phone: 404-639-3091). Also, we do not claim that “no-one has ever been infected with prion disease from eating venison.” Our conclusion stating that we found no strong evidence of CWD transmission to humans in the article you quoted or in any other forum is limited to the patients we investigated.
Ermias Belay, M.D.
Centers for Disease Control and Prevention
Sent: Sunday, September 29, 2002 10:15 AM
To: firstname.lastname@example.org; email@example.com; ebb8@CDC.GOV
Subject: TO CDC AND NIH – PUB MED- 3 MORE DEATHS – CWD – YOUNG
Sunday, November 10, 2002 6:26 PM ……snip……..end…………..TSS
Exotic Meats USA Announces Urgent Statewide Recall of Elk Tenderloin Because It May Contain Meat Derived From An Elk Confirmed To Have CWD
Cross-sequence transmission of sporadic Creutzfeldt-Jakob disease creates a new prion strain
Date: August 25, 2007 at 12:42 pm PST
our results raise the possibility that CJD cases classified as VV1 may include cases caused by iatrogenic transmission of sCJD-MM1 prions or food-borne infection by type 1 prions from animals, e.g., chronic wasting disease prions in cervid. In fact, two CJD-VV1 patients who hunted deer or consumed venison have been reported (40, 41). The results of the present study emphasize the need for traceback studies and careful re-examination of the biochemical properties of sCJD-VV1 prions.
Clearly, it is premature to draw firm conclusions about CWD passing naturally into humans, cattle and sheep, but the present results suggest that CWD transmissions to humans would be as limited by PrP incompatibility as transmissions of BSE or sheep scrapie to humans. Although there is no evidence that sheep scrapie has affected humans, it is likely that BSE has caused variant CJD in 74 people (definite and probable variant CJD cases to date according to the UK CJD Surveillance Unit). Given the presumably large number of people exposed to BSE infectivity, the susceptibility of humans may still be very low compared with cattle, which would be consistent with the relatively inefficient conversion of human PrP-sen by PrPBSE. Nonetheless, since humans have apparently been infected by BSE, it would seem prudent to take reasonable measures to limit exposure of humans (as well as sheep and cattle) to CWD infectivity as has been recommended for other animal TSEs.