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CWD Infected buck found 40 miles from Michigan's U.P.

Posted Nov 13 2010 2:45pm
Posted: 10:22 a.m. Nov. 13, 2010

Infected buck found 40 miles from Michigan's U.P.


Comments (15) Recommend Print E-mail Letter to the editor Share Facebook Twitter FarkIt Digg Reddit Newsvine Buzz up!A whitetail buck infected with chronic wasting disease has been found on a Wisconsin shooting preserve 40 miles from Michigan’s Upper Peninsula. That will trigger a ban on baiting and feeding deer in the UP, although the DNRE said it won’t affect the firearms deer season that opens Monday.

Mary Dettloff, a spokesperson the Department of Natural Resources and Environment, said that it’s too late to stop people from hunting over bait in the UP because so much corn, beets and other lures have already been put out. But the agency will ask hunters to stop putting out more.

Baiting already is banned in the Lower Peninsula, where a doe on a Kent County deer ranch was found to have the disease two years ago.

CWD is an invariably fatal disease of deer and elk that was first diagnosed in Colorado about 40 years ago. Wildlife biologists say that it is spread by deer coming in contact with mucous and urine from infected deer and that concentrating deer nose-to-nose at bait piles increases the chances of infection.

Dettloff said the DNRE was informed about the Wisconsin buck Friday but would not act on the discovery until it is confirmed by a “gold standard” test at an animal disease laboratory at Ames, Iowa (usually within a few weeks).

If it CWD is confirmed in the Ashland deer, the existing protocol the agency is required to ban baiting and recreational feeding of deer in the UP.

The protocol says, “In the event CWD is documented within Michigan or within 50 miles of Michigan’s border with another state or Canadian province, the (MDNRE) Director shall issue an interim order banning the use of bait and banning the feeding of deer and elk within the peninsula adjacent to the adjoining state or province with CWD or containing CWD.”

While baiting is banned in the Lower Peninsula, the law is very unpopular with many hunters and has been openly flouted. Corn, beets and carrots advertised as deer bait are stacked on pallets at stores and gas stations throughout the state, and bait dealers say hunters have been buying nearly as much as they did before the ban went into effect.

Contact ERIC SHARP: 313-222-2511 or Read more in his outdoors blog at Order his book "Fishing Michigan" for $15.95 at or by calling 800-245-5082.

Read more: Infected buck found 40 miles from Michigan's U.P. Detroit Free Press -

CWD find raises threat up north Preliminary test shows ill deer at shooting preserve near Ashland By Paul A. Smith and Lee Bergquist of the Journal Sentinel

Nov. 12, 2010 (7) Comments

A white-tailed deer from a shooting preserve in northern Wisconsin has tested positive on a screening test for chronic wasting disease - a discovery that has the potential to bring the disease to a new part of the state.

Complicating the situation: An inspection of the fence at the 900-acre preserve in late October showed signs of damage, raising the possibility that deer could have escaped from the facility, a Department of Natural Resources official said Friday.

"The test and the fence issues certainly are a concern to the DNR," said Davin Lopez, chronic wasting disease coordinator for the agency.

The CWD-positive result was first identified in a test at the Wisconsin Veterinary Diagnostic Laboratory in Madison, according to the Wisconsin Department of Agriculture, Trade and Consumer Protection.

Tissue samples from the deer, reportedly a 3-year-old buck on a shooting preserve near Ashland in Bayfield County, have been sent to the Veterinary Diagnostic Laboratory in Ames, Iowa.

"We want to make sure it's not a false positive before making any formal announcements," said Lee Sensenbrenner, spokesman for the agriculture department.

The Ames lab at Iowa State University runs the "gold standard" test for CWD

Final results will not be known until Wednesday or later, said another agriculture spokeswoman, Donna Gilson.

Pending confirmation, the department declined to release the name of the shooting preserve.

If confirmed, the finding would be the first CWD-positive deer in northern Wisconsin. The fatal deer disease has been found in wild deer in a 1,000-square-mile Chronic Wasting Disease Management Zone in southern Wisconsin as well as game farms in other parts of the state. Two facilities in Portage County, one in Crawford County and another in Manitowoc County have had positive reports of CWD outside the state's disease management zone.

The disease was first discovered in Wisconsin in 2002.

"Not good," said Mike Riggle, a veterinarian, hunter and Wisconsin Conservation Congress member from Medford, of the latest development. "Everybody has been holding their breath for the last eight years."

"Folks in the north had thought that this isn't our problem," said Riggle, who also serves as chairman of the CWD Committee of the Wisconsin Conservation Congress. "Now, boom, it is our problem."

Riggle said the likelihood of a false-positive was "very, very remote."

The buck was tested as part of a protocol on deer farms in the state. The state agriculture department has authority over deer farms in Wisconsin.

The DNR has begun contingency planning, said Tom Hauge, wildlife chief, including possibly increased testing of wild deer in the area.

Deer shot in the area during this year's hunting season will be tested for the disease.

Previous testing of over 1,000 wild deer in the Ashland area had turned up no CWD-positives, according to the DNR.

Though fatal to deer and elk, CWD has shown no link to human health or livestock.

Also, a positive test doesn't necessarily expose wild deer in the area to the disease.

A game farm near Almond in Portage County produced 82 infected deer. Most were killed in 2006, and the rash of positives represented the highest infection rate ever reported in the United States, the DNR said.

Before the shooting, officials discovered that a hole had been cut in the fence and that the captive bucks - about 30 - had escaped and were never found.

But state records show that analyses of 2,559 wild deer from Portage and Waushara counties have shown no signs of the disease.



Friday, February 20, 2009

Both Sides of the Fence: A Strategic Review of Chronic Wasting Disease

Saturday, September 06, 2008

Chronic wasting disease in a Wisconsin white-tailed deer farm 79% INFECTION RATE

Contents: September 1 2008, Volume 20, Issue 5

snip...see full text ;

Tuesday, February 09, 2010

Chronic Wasting Disease: Surveillance Update North America: February 2010


>>> In addition, we documented horizontal transmission of CWD from inoculated mice and to un-inoculated cohabitant cage-mates. <<<

disturbing. ...TSS

Sunday, December 06, 2009

Detection of Sub-Clinical CWD Infection in Conventional Test-Negative Deer Long after Oral Exposure to Urine and Feces from CWD+ Deer

Wednesday, March 18, 2009

Detection of CWD Prions in Urine and Saliva of Deer by Transgenic Mouse Bioassay

Tuesday, June 16, 2009

Infectious Prions in Pre-Clinical Deer and Transmission of Chronic Wasting Disease Solely by Environmental Exposure

Wednesday, October 14, 2009

Detection of protease-resistant cervid prion protein in water from a CWD-endemic area


Sunday, November 01, 2009

American crows (Corvus brachyrhynchos) and potential spreading of CWD through feces of digested infectious carcases

Wednesday, January 07, 2009

CWD to tighten taxidermy rules Hunters need to understand regulations

Wednesday, September 08, 2010




Chad Johnson1, Judd Aiken2,3,4 and Debbie McKenzie4,5 1 Department of Comparative Biosciences, University of Wisconsin, Madison WI, USA 53706 2 Department of Agriculture, Food and Nutritional Sciences, 3 Alberta Veterinary Research Institute, 4.Center for Prions and Protein Folding Diseases, 5 Department of Biological Sciences, University of Alberta, Edmonton AB, Canada T6G 2P5 The identification and characterization of prion strains is increasingly important for the diagnosis and biological definition of these infectious pathogens. Although well-established in scrapie and, more recently, in BSE, comparatively little is known about the possibility of prion strains in chronic wasting disease (CWD), a disease affecting free ranging and captive cervids, primarily in North America. We have identified prion protein variants in the white-tailed deer population and demonstrated that Prnp genotype affects the susceptibility/disease progression of white-tailed deer to CWD agent. The existence of cervid prion protein variants raises the likelihood of distinct CWD strains. Small rodent models are a useful means of identifying prion strains. We intracerebrally inoculated hamsters with brain homogenates and phosphotungstate concentrated preparations from CWD positive hunter-harvested (Wisconsin CWD endemic area) and experimentally infected deer of known Prnp genotypes. These transmission studies resulted in clinical presentation in primary passage of concentrated CWD prions. Subclinical infection was established with the other primary passages based on the detection of PrPCWD in the brains of hamsters and the successful disease transmission upon second passage. Second and third passage data, when compared to transmission studies using different CWD inocula (Raymond et al., 2007) indicate that the CWD agent present in the Wisconsin white-tailed deer population is different than the strain(s) present in elk, mule-deer and white-tailed deer from the western United States endemic region.

Potential Venison Exposure Among FoodNet Population Survey Respondents, 2006-2007

Ryan A. Maddox1*, Joseph Y. Abrams1, Robert C. Holman1, Lawrence B. Schonberger1, Ermias D. Belay1 Division of Viral and Rickettsial Diseases, National Center for Zoonotic, Vector-Borne, and Enteric Diseases, Centers for Disease Control and Prevention, Atlanta, GA *Corresponding author e-mail:

The foodborne transmission of bovine spongiform encephalopathy to humans, resulting in variant Creutzfeldt-Jakob disease, indicates that humans can be susceptible to animal prion diseases. However, it is not known whether foodborne exposure to the agent causing chronic wasting disease (CWD) in cervids can cause human disease. The United States Foodborne Diseases Active Surveillance Network (FoodNet) conducts surveillance for foodborne diseases through an extensive survey administered to respondents in selected states. To describe the frequency of deer and elk hunting and venison consumption, five questions were included in the 2006-2007 FoodNet survey. This survey included 17,372 respondents in ten states: California, Colorado, Connecticut, Georgia, Maryland, Minnesota, New Mexico, New York, Oregon, and Tennessee. Of these respondents, 3,220 (18.5%) reported ever hunting deer or elk, with 217 (1.3%) reporting hunting in a CWD-endemic area (northeastern Colorado, southeastern Wyoming, and southwestern Nebraska). Of the 217 CWD-endemic area hunters, 74 (34.1%) were residents of Colorado. Respondents reporting hunting were significantly more likely to be male than female (prevalence ratio: 3.3, 95% confidence interval: 3.1-3.6) and, in general, older respondents were significantly more likely to report hunting than younger respondents. Venison consumption was reported by more than half (67.4%) of the study population, and most venison consumers (94.1%) reported that at least half of their venison came from the wild. However, more than half (59.1%) of the consumers reported eating venison only one to five times in their life or only once or twice a year. These findings indicate that a high percentage of the United States population engages in hunting and/or venison consumption. If CWD continues to spread to more areas across the country, a substantial number of people could potentially be exposed to the infectious agent.

Sunday, April 12, 2009

CWD UPDATE Infection Studies in Two Species of Non-Human Primates and one Environmental reservoir infectivity study and evidence of two strains

I will quote what one of the leading scientist in the world on prion disease once said about CWD, and then post some data on this topic. Good question, one that should be addressed. I will attempt to address it. I am a meat eater, and I don't care what you and others eat, but on the topic of human and animal Transmissible Spongiform Encephalopathy, I can assure you that you have not been told the complete truth. This is a long post, take the information, and please look at the source of the data. I post this data on blogs, no advertisements, not making money doing this. The information is for your benefit, please use as you wish. ...kind regards, terry

A. Aguzzi - Chronic Wasting Disease (CWD) also needs to be addressed.

Most serious because of rapid horizontal spread and higher prevalence than BSE in UK, up to 15% in some populations.

Also may be a risk to humans - evidence that it is not dangerous to humans is thin. ...end

REMEMBER, there are over twenty strains of typical scrapie, and the atypical scrapie cases (Nor-98) are mounting, two documented strains of Transmissible Mink Encephalopathy, and 3 strains of BSE in cattle, all of which have been documented in North America, all have been shown to transmit to man via lab studies in mice, with the L-type atypical BSE being much more virulent. Also, now there are two strains of CWD. all these TSE in different animals have been rendered and put into feed for livestock producing animals for human and animal in the USA. This is fact.

can humans get a prion disease from deer or elk that have CWD ?

as someone else stated here, no one knows for sure to date, but the likelyhood of being exposed to CWD from eating a CWD deer or elk is high, and then years on down the road, regardless whether or not this person ever goes clinical, due to any medical surgical, or dental procedures, blood donations, the agent will continue to spread, more humans exposed. the fact that some of these atypical TSE are mutating and becoming more virulent is very troublesome. the incubation time is shortened.

I assure you that the FDA did not recall this CWD postive elk meat in commerce, for the well being of the dead CWD postive elk ;

Wednesday, March 18, 2009

Noah's Ark Holding, LLC, Dawson, MN RECALL Elk products contain meat derived from an elk confirmed to have CWD NV, CA, TX, CO, NY, UT, FL, OK RECALLS AND FIELD CORRECTIONS: FOODS CLASS II

please see ;



PRODUCT a) Elk Meat, Elk Tenderloin, Frozen in plastic vacuum packaging. Each package is approximately 2 lbs., and each case is approximately 16 lbs.; Item number 755125, Recall # F-129-9;

b) Elk Meat, Elk Trim, Frozen; Item number 755155, Recall # F-130-9;

c) Elk Meat, French Rack, Chilled. Item number 755132, Recall # F-131-9;

d) Elk Meat, Nude Denver Leg. Item number 755122, Recall # F-132-9;

e) Elk Meat, New York Strip Steak, Chilled. Item number 755128, Recall # F-133-9;

f) Elk Meat, Flank Steak Frozen. Item number 755131, Recall # F-134-9; CODE Elk Meats with production dates of December 29, 30, and 31 RECALLING FIRM/MANUFACTURER Recalling Firm: Sierra Meats, Reno, NV, by telephone on January 29, 2009 and press release on February 9, 2009. Manufacturer: Noah’s Ark Holding, LLC, Dawson, MN. Firm initiated recall is ongoing. REASON Elk products contain meat derived from an elk confirmed to have Chronic Wasting Disease (CWD). VOLUME OF PRODUCT IN COMMERCE Unknown DISTRIBUTION NV, CA, TX, CO, NY, UT, FL, OK




Sunday, April 12, 2009

CWD UPDATE Infection Studies in Two Species of Non-Human Primates and one Environmental reservoir infectivity study and evidence of two strains

Thursday, April 03, 2008

A prion disease of cervids: Chronic wasting disease

2008 1: Vet Res. 2008 Apr 3;39(4):41

A prion disease of cervids: Chronic wasting disease

Sigurdson CJ.


*** twenty-seven CJD patients who regularly consumed venison were reported to the Surveillance Center***,


full text ;

From: TSS (


Date: September 30, 2002 at 7:06 am PST

From: "Belay, Ermias"

Cc: "Race, Richard (NIH)" ; ; "Belay,


Sent: Monday, September 30, 2002 9:22 AM


Dear Sir/Madam,

In the Archives of Neurology you quoted (the abstract of which was

attached to your email), we did not say CWD in humans will present like

variant CJD.

That assumption would be wrong. I encourage you to read the whole

article and call me if you have questions or need more clarification

(phone: 404-639-3091). Also, we do not claim that "no-one has ever been

infected with prion disease from eating venison." Our conclusion stating

that we found no strong evidence of CWD transmission to humans in the

article you quoted or in any other forum is limited to the patients we


Ermias Belay, M.D.

Centers for Disease Control and Prevention

-----Original Message-----

Sent: Sunday, September 29, 2002 10:15 AM

To:;; ebb8@CDC.GOV



Sunday, November 10, 2002 6:26 PM ......snip........end..............TSS

SEE also ;

A. Aguzzi - Chronic Wasting Disease (CWD) also needs to be addressed. Most

serious because of rapid horizontal spread and higher prevalence than BSE in

UK, up to 15% in some populations. Also may be a risk to humans - evidence

that it is not dangerous to humans is thin.


Chronic Wasting Disease and Potential Transmission to Humans

Ermias D. Belay,* Ryan A. Maddox,* Elizabeth S. Williams,? Michael W. Miller,? Pierluigi Gambetti,§ and Lawrence B. Schonberger*

*Centers for Disease Control and Prevention, Atlanta, Georgia, USA; ?University of Wyoming, Laramie, Wyoming, USA; ?Colorado Division of Wildlife, Fort Collins, Colorado, USA; and §Case Western Reserve University, Cleveland, Ohio, USA

Suggested citation for this article: Belay ED, Maddox RA, Williams ES, Miller MW, Gambetti P, Schonberger LB. Chronic wasting disease and potential transmission to humans. Emerg Infect Dis [serial on the Internet]. 2004 Jun [date cited]. Available from


Chronic wasting disease (CWD) of deer and elk is endemic in a tri-corner area of Colorado, Wyoming, and Nebraska, and new foci of CWD have been detected in other parts of the United States. Although detection in some areas may be related to increased surveillance, introduction of CWD due to translocation or natural migration of animals may account for some new foci of infection. Increasing spread of CWD has raised concerns about the potential for increasing human exposure to the CWD agent. The foodborne transmission of bovine spongiform encephalopathy to humans indicates that the species barrier may not completely protect humans from animal prion diseases. Conversion of human prion protein by CWD-associated prions has been demonstrated in an in vitro cell-free experiment, but limited investigations have not identified strong evidence for CWD transmission to humans. More epidemiologic and laboratory studies are needed to monitor the possibility of such transmissions.

snip...full text ;

Volume 12, Number 10-October 2006


Human Prion Disease and Relative Risk Associated with Chronic Wasting Disease

Samantha MaWhinney,* W. John Pape,? Jeri E. Forster,* C. Alan Anderson,?§ Patrick Bosque,?¶ and Michael W. Miller#

*University of Colorado at Denver and Health Sciences Center, Denver, Colorado, USA; ?Colorado Department of Public Health and Environment, Denver, Colorado, USA; ?University of Colorado School of Medicine, Denver, Colorado, USA; §Denver Veteran's Affairs Medical Center, Denver, Colorado, USA; ¶Denver Health Medical Center, Denver, Colorado, USA; and #Colorado Division of Wildlife, Fort Collins, Colorado, USA

Suggested citation for this article

The transmission of the prion disease bovine spongiform encephalopathy (BSE) to humans raises concern about chronic wasting disease (CWD), a prion disease of deer and elk. In 7 Colorado counties with high CWD prevalence, 75% of state hunting licenses are issued locally, which suggests that residents consume most regionally harvested game. We used Colorado death certificate data from 1979 through 2001 to evaluate rates of death from the human prion disease Creutzfeldt-Jakob disease (CJD). The relative risk (RR) of CJD for CWD-endemic county residents was not significantly increased (RR 0.81, 95% confidence interval [CI] 0.40-1.63), and the rate of CJD did not increase over time (5-year RR 0.92, 95% CI 0.73-1.16). In Colorado, human prion disease resulting from CWD exposure is rare or nonexistent. However, given uncertainties about the incubation period, exposure, and clinical presentation, the possibility that the CWD agent might cause human disease cannot be eliminated.

snip... full text ;

full text ;

(10) Transmission of Elk and Deer Prions to Transgenic Mice

(30) Neurobiology of Disease Chronic Wasting Disease of Elk: Transmissibility to Humans Examined by Transgenic Mouse Models

(13) Prions in Skeletal Muscles of Deer with Chronic Wasting Disease;311/5764/1117

(14) Volume 15, Number 5–May 2009 Research Chronic Wasting Disease Prions in Elk Antler Velvet

Friday, May 14, 2010

Prion Strain Mutation Determined by Prion Protein Conformational Compatibility and Primary Structure

Published Online May 13, 2010 Science DOI: 10.1126/science.1187107 Science Express Index

In Confidence - Perceptions of unconventional slow virus diseases of animals in the USA - APRIL-MAY 1989 - G A H Wells

3. Prof. A Robertson gave a brief account of BSE. The US approach was to accord it a very low profile indeed. Dr. A Thiermann showed the picture in the ''Independent'' with cattle being incinerated and thought this was a fanatical incident to be avoided in the US at all costs. BSE was not reported in the USA.


CWD occurred principally in two locations, this one at Sybille and in a similar faccility at Fort Collins, Colorado, some 120 miles southwest. It was estimated that in total probably 60-70 cases of CWD have occurred.

It was difficult to gain a clear account of incidence and temporal sequence of events (-this presumably is data awaiting publication - see below) but during the period 1981-1984, 10-15 cases occurred at the Sybille facility.

The moribidity amongst mule deer in the facilities ie. those of the natural potentially exposed group has been about 90% with 100% mortality.


Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.

see full text 33 pages ;


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