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ACA Council Meets to Endorse Several Proposed USAHA Resolutions (CWD TSE PRION DISEASE)

Posted Oct 19 2013 3:23pm

 


Resolutions to Reform CWD Standards, TB, Brucellosis, and ACA Seeking USAHA Board Seat

The American Cervid Alliance Leadership Council met Thursday, October 17, 2013, to discuss possible opportunities at the upcoming United States Animal Health Association (USAHA) Conference this weekend in San Diego. The ACA’s Moderator, Eric Mohlman, wanted all the alliance council members to have an understanding of the agenda effecting our industry at USAHA prior to the leader’s arrival.  

Several ideas were discussed including how to strengthen the cervid industry’s voice within USAHA and ways to decrease on-farm testing requirements and general regulations.  Several of these ideas will be put forth, during the conference, in the form of resolutions and presented to the Brucellosis, Tuberculosis, and Captive Wildlife and Alternative Livestock Committees.

Eric Mohlman advised the Alliance that he has had several discussions with key USAHA leaders about the possibly of approving the ACA as an allied organization and board member of USAHA. Currently, three of the national cervid associations, which include the North American Elk Breeders Association, North American Deer Farmers Association, and Exotic Wildlife Association, serve on the board of directors.  The possibility of the ACA having its own seat, as an allied organization representing thirty cervid associations, would increase the presence of the industry voice on the national level.  A motion was made by Kim Kafka of the North American Elk Breeders Association, seconded by Brian Wagner of the Minnesota Elk Breeders Association, to approve the ACA moving forward to seek the USAHA Board seat.  The motion was adopted unanimously, 22-0.  The Alliance nominated two council members to serve as the interim representative for the ACA, if approved.  The nominees were Eric Mohlman and Laurie Seale. After the roll call vote, Laurie Seale was approved by a vote of 13-7.  Next year the ACA will conduct a nominating vetting process for the 2014 USAHA Conference representative.  
Charly Seale, of the Exotic Wildlife Association, stated that many of the cervid industry’s issues have, in many instances, been minimized and lost in the all encompassing Captive Wildlife and Alternative Livestock Committee. Seale said that committee discusses everything from wild animals to elephants and the cervid industry needs its own committee to focus solely on cervid farming issues.  Seale has been invited to speak with the USAHA Executive Committee this weekend to propose a new and separate farmed cervid committee, which, if approved would begin at the 2014 conference.  A motion was made, by Tim Condict of the Deer Breeders Corporation, for the ACA Council to endorse the resolution. Curt Waldvogel of the Second Ark Foundation, seconded the motion. The motion was adopted 22-0.  
Two resolutions were presented to the council, which will ask USAHA to urge the USDA to withdraw the current existing CWD standards document versions 1 through 22 and also ask for a new CWD Standards Working Group to be created with cervid industry, state agriculture, and USDA representatives, without wildlife officials, to redraft a new standards documents and remove the old Version 1 document from their policy. Seale stated, “We tried improving the Chronic Wasting Disease program standards document 22 times over a sixth month period and it is not even close. Version 1( July 2012), which according to USDA/ APHIS, the industry is operating  under needs to  be completely withdrawn because some states are already using it and even Dr Klein admits was not their best effort.”  A motion was made by Todd Landt, of the Iowa Whitetail Deer Association, to ask the ACA to endorse the two resolutions when presented to the Captive Wildlife and Alternative Livestock Committee. Kim Kafka of the North American Elk Breeders Association seconded the motion.   The motion was adopted 18-0, with one association passing. 
Two additional resolutions will be presented in the Brucellosis and Tuberculosis Committees to ask for longer testing intervals for TB reaccreditation and Brucellosis recertification.  Two separate motions were offered by Michael Heiter of the Pennsylvania Deer Farmers Association and Joel Espe of the Wisconsin Commercial Deer and Elk Farmers Association, respectively, for the ACA endorsement.  Both motions were unanimously adopted for both resolutions. 
ACA Moderator Eric Mohlman thanked the council for their input on the proposed resolutions.  Mohlman, said, “Last year at USAHA, the association leaders didn’t know what the others within the industry were planning.  Now all the thirty associations participating with the ACA are all up to date on industry proposals before we get to the convention. We are heading to San Diego as a team and united as an industry.”
 
http://www.americancervidalliance.org/news.cfm?id=68
 
IT seems the USAHA is going to cater to the cervid industry now, and I pray that it does not wind up being a broken industry fed bunch like the OIE/USDA, but things have been looking that way recently in regards with animal TSE prion disease. seems nothing has been science based of late, in my opinion. heck, from the looks of it ;
 
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Two resolutions were presented to the council, which will ask USAHA to urge the USDA to withdraw the current existing CWD standards document versions 1 through 22 and also ask for a new CWD Standards Working Group to be created with cervid industry, state agriculture, and USDA representatives, without wildlife officials, to redraft a new standards documents and remove the old Version 1 document from their policy. Seale stated, “We tried improving the Chronic Wasting Disease program standards document 22 times over a sixth month period and it is not even close. Version 1( July 2012), which according to USDA/ APHIS, the industry is operating  under needs to  be completely withdrawn because some states are already using it and even Dr Klein admits was not their best effort.”  A motion was made by Todd Landt, of the Iowa Whitetail Deer Association, to ask the ACA to endorse the two resolutions when presented to the Captive Wildlife and Alternative Livestock Committee. Kim Kafka of the North American Elk Breeders Association seconded the motion.   The motion was adopted 18-0, with one association passing.  ...
 
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hell, the USAHA should just step aside and let the shooting pens write their own rules, it’s all big ag now.
seems with the USDA all aboard now with the captives now being livestock, it’s all over but the crying, the wild as we knew it, and wild cervids there from, do not have a chance now, a sad day for hunters in my opinion. ...tss
 
 
USDA TO PGC ONCE CAPTIVES ESCAPE "it‘s no longer its business.”

 

 

Sunday, January 06, 2013

 

USDA TO PGC ONCE CAPTIVES ESCAPE "it‘s no longer its business.”

 


 

 

 Tuesday, October 15, 2013

 

Indiana State to appeal high-fence deer hunting ruling

 


 

 
Thursday, October 03, 2013

TAHC ADOPTS CWD RULE THAT the amendments ''remove'' the requirement for a specific fence height for captives Texas Animal Health Commission (TAHC) ANNOUNCEMENT

October 3, 2013


http://chronic-wasting-disease.blogspot.com/2013/10/tahc-adopts-cwd-rule-that-amendments.html

 

 

Wednesday, October 16, 2013

 

Pennsylvania Adjusts CWD Rules

 


 

 

 PRION2013 CONGRESSIONAL ABSTRACTS CWD

 

 

Thursday, August 08, 2013

 

Characterization of the first case of naturally occurring chronic wasting disease in a captive red deer (Cervus elaphus) in North America

 


 

 

 

Sunday, September 01, 2013

 

hunting over gut piles and CWD TSE prion disease

 


 

 

 

Wednesday, September 04, 2013

 

***cwd - cervid captive livestock escapes, loose and on the run in the wild...

 


 

 

 

Monday, June 24, 2013

 

The Effects of Chronic Wasting Disease on the Pennsylvania Cervid Industry Following its Discovery

 


 

 

 

Thursday, July 11, 2013

 

The New Hornographers: The Fight Over the Future of Texas Deer, Captive shooting pens, and the CWD TSE prion disease

 


 

 

 

Tuesday, July 02, 2013

 

National Rifle Association and the Unified Sportsman of Florida support a Florida ban on the importation of captive deer and cervids into Florida

 


 

 

 

Sunday, June 09, 2013

 

Missouri House forms 13-member Interim Committee on the Cause and Spread of Chronic Wasting Disease CWD

 


 

 

 

Tuesday, April 16, 2013

 

Cervid Industry Unites To Set Direction for CWD Reform and seem to ignore their ignorance and denial in their role in spreading Chronic Wasting Disease

 


 

 

 

Monday, October 07, 2013

 

The importance of localized culling in stabilizing chronic wasting disease prevalence in white-tailed deer populations

 


 

 

 

Saturday, February 04, 2012

 

Wisconsin 16 MONTH age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised

 


 

 
how many states have $465,000., and can quarantine and purchase there from, each cwd said infected farm, but how many states can afford this for all the cwd infected cervid game ranch type farms ??

 

 

Tuesday, December 20, 2011

 

CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011

 

The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.

 

RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.

 

 

SUMMARY:

 

 


 

 

 

recently, a report came out in the U.K., about risk factors from entry of CWD from the USA. I think you might find interest there ;

 

 

Friday, December 14, 2012

 

DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012

 

snip...

 

In the USA, under the Food and Drug Administration’s BSE Feed Regulation (21 CFR 589.2000) most material (exceptions include milk, tallow, and gelatin) from deer and elk is prohibited for use in feed for ruminant animals. With regards to feed for non-ruminant animals, under FDA law, CWD positive deer may not be used for any animal feed or feed ingredients. For elk and deer considered at high risk for CWD, the FDA recommends that these animals do not enter the animal feed system. However, this recommendation is guidance and not a requirement by law.

 

Animals considered at high risk for CWD include:

 

1) animals from areas declared to be endemic for CWD and/or to be CWD eradication zones and

 

2) deer and elk that at some time during the 60-month period prior to slaughter were in a captive herd that contained a CWD-positive animal.

 

Therefore, in the USA, materials from cervids other than CWD positive animals may be used in animal feed and feed ingredients for non-ruminants.

 

The amount of animal PAP that is of deer and/or elk origin imported from the USA to GB can not be determined, however, as it is not specified in TRACES. It may constitute a small percentage of the 8412 kilos of non-fish origin processed animal proteins that were imported from US into GB in 2011.

 

Overall, therefore, it is considered there is a __greater than negligible risk___ that (nonruminant) animal feed and pet food containing deer and/or elk protein is imported into GB.

 

There is uncertainty associated with this estimate given the lack of data on the amount of deer and/or elk protein possibly being imported in these products.

 

snip...

 

36% in 2007 (Almberg et al., 2011). In such areas, population declines of deer of up to 30 to 50% have been observed (Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as 30% (EFSA, 2011). The clinical signs of CWD in affected adults are weight loss and behavioural changes that can span weeks or months (Williams, 2005). In addition, signs might include excessive salivation, behavioural alterations including a fixed stare and changes in interaction with other animals in the herd, and an altered stance (Williams, 2005). These signs are indistinguishable from cervids experimentally infected with bovine spongiform encephalopathy (BSE). Given this, if CWD was to be introduced into countries with BSE such as GB, for example, infected deer populations would need to be tested to differentiate if they were infected with CWD or BSE to minimise the risk of BSE entering the human food-chain via affected venison.

 

snip...

 

The rate of transmission of CWD has been reported to be as high as 30% and can approach 100% among captive animals in endemic areas (Safar et al., 2008).

 

snip...

 

In summary, in endemic areas, there is a medium probability that the soil and surrounding environment is contaminated with CWD prions and in a bioavailable form. In rural areas where CWD has not been reported and deer are present, there is a greater than negligible risk the soil is contaminated with CWD prion.

 

snip...

 

In summary, given the volume of tourists, hunters and servicemen moving between GB and North America, the probability of at least one person travelling to/from a CWD affected area and, in doing so, contaminating their clothing, footwear and/or equipment prior to arriving in GB is greater than negligible. For deer hunters, specifically, the risk is likely to be greater given the increased contact with deer and their environment. However, there is significant uncertainty associated with these estimates.

 

snip...

 

Therefore, it is considered that farmed and park deer may have a higher probability of exposure to CWD transferred to the environment than wild deer given the restricted habitat range and higher frequency of contact with tourists and returning GB residents.

 

snip...

 

 


 

 

 

SNIP...SEE ;

 

 

Friday, December 14, 2012

 

DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012

 

 


 

 

 

Wednesday, September 25, 2013

 

Inspections, Compliance, Enforcement, and Criminal Investigations BSE TSE PRION 2013

 


 

 

 

DOCKET-- 03D-0186 -- FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

 

Date: Fri, 16 May 2003 11:47:37 –0500

 

EMC 1 Terry S. Singeltary Sr. Vol #: 1

 


 


 

 

 

PLEASE SEE FULL TEXT SUBMISSION ;

 

 


 

 

 

please see more on the potential of the prion uptake from plants, and see what SEAC said about the one study that showed prion uptake of the Tomato plant, although the test was never repeated ;

 

 

Furthermore, an unpublished study had indicated low level absorption of PrP from soil by tomato plants although it should be noted that this study had not been repeated. Details of this work would be sent to the SEAC Secretary. Dr Matthews explained that most of the manure from animals challenged with high doses of BSE had already been composted and used for coppicing. Members agreed that the risks from disposal of residual manure from experimental animals would be much less than historic risks of on farm contamination from naturally infected animals at the height of the BSE epidemic. ...SNIP...END

 

Uptake of Prions into Plants

 


 

 

 

Prion2013

 

Friday, August 09, 2013

 

***CWD TSE prion, plants, vegetables, and the potential for environmental contamination

 


 

 

 

 

PRION2013 CONGRESSIONAL ABSTRACTS CWD

 

 

Sunday, August 25, 2013

 

HD.13: CWD infection in the spleen of humanized transgenic mice

 

Liuting Qing and Qingzhong Kong

 

Case Western Reserve University; Cleveland, OH USA

 

Chronic wasting disease (CWD) is a widespread prion disease in free-ranging and captive cervid species in North America, and there is evidence suggesting the existence of multiple CWD strains. The susceptibility of human CNS and peripheral organs to the various CWD prion strains remains largely unclear. Current literature suggests that the classical CWD strain is unlikely to infect human brain, but the potential for peripheral infection by CWD in humans is unknown. We detected protease-resistant PrpSc in the spleens of a few humanized transgenic mice that were intracerebrally inoculated with natural CWD isolates, but PrpSc was not detected in the brains of any of the CWD-inoculated mice. Our ongoing bioassays in humanized Tg mice indicate that intracerebral challenge with such PrpSc-positive humanized mouse spleen already led to prion disease in most animals.

 

***These results indicate that the CWD prion may have the potential to infect human peripheral lymphoid tissues.

 

 

Oral.15: Molecular barriers to zoonotic prion transmission: Comparison of the ability of sheep, cattle and deer prion disease isolates to convert normal human prion protein to its pathological isoform in a cell-free system

 

Marcelo A.Barria,1 Aru Balachandran,2 Masanori Morita,3 Tetsuyuki Kitamoto,4 Rona Barron,5 Jean Manson,5 Richard Kniqht,1 James W. lronside1 and Mark W. Head1

 

1National CJD Research and Surveillance Unit; Centre for Clinical Brain Sciences; School of Clinical Sciences; The University of Edinburgh; Edinburgh, UK; 2National and OIE Reference Laboratory for Scrapie and CWD; Canadian Food Inspection Agency; Ottawa Laboratory; Fallowfield. ON Canada; 3Infectious Pathogen Research Section; Central Research Laboratory; Japan Blood Products Organization; Kobe, Japan; 4Department of Neurological Science; Tohoku University Graduate School of Medicine; Sendai. Japan; 5Neurobiology Division; The Roslin Institute and R(D)SVS; University of Edinburgh; Easter Bush; Midlothian; Edinburgh, UK

 

Background. Bovine spongiform encephalopathy (BSE) is a known zoonotic prion disease, resulting in variant Creurzfeldt- Jakob disease (vCJD) in humans. In contrast, classical scrapie in sheep is thought to offer little or no danger to human health. However, a widening range of prion diseases have been recognized in cattle, sheep and deer. The risks posed by individual animal prion diseases to human health cannot be determined a priori and are difficult to assess empirically. The fundamemal event in prion disease pathogenesis is thought to be the seeded conversion of normal prion protein (PrPC) to its pathological isoform (PrPSc). Here we report the use of a rapid molecular conversion assay to test whether brain specimens from different animal prion diseases are capable of seeding the conversion of human PrPC ro PrPSc.

 

Material and Methods. Classical BSE (C-type BSE), H-type BSE, L-type BSE, classical scrapie, atypical scrapie, chronic wasting disease and vCJD brain homogenates were tested for their ability to seed conversion of human PrPC to PrPSc in protein misfolding cyclic amplification (PMCA) reactions. Newly formed human PrPSc was detected by protease digestion and western blotting using the antibody 3F4.

 

Results. C-type BSE and vCJD were found to efficiently convert PrPC to PrPSc. Scrapie failed to convert human PrPC to PrPSc. Of the other animal prion diseases tested only chronic wasting disease appeared to have the capability ro convert human PrPC to PrPSc. The results were consistent whether the human PrPC came from human brain, humanised transgenic mouse brain or from cultured human cells and the effect was more pronounced for PrPC with methionine at codon 129 compared with that with valine.

 

Conclusion. Our results show that none of the tested animal prion disease isolates are as efficient as C-type BSE and vCJD in converting human prion protein in this in vitro assay.

 

 

***However, they also show that there is no absolute barrier ro conversion of human prion protein in the case of chronic wasting disease.

 

 

 

PRION2013 CONGRESSIONAL ABSTRACTS CWD

 

 

Sunday, August 25, 2013

 

***Chronic Wasting Disease CWD risk factors, humans, domestic cats, blood, and mother to offspring transmission

 


 

 

 

Sunday, July 21, 2013

 

*** As Chronic Wasting Disease CWD rises in deer herd, what about risk for humans?

 


 

 

 

Thursday, October 10, 2013

 

CJD REPORT 1994 increased risk for consumption of veal and venison and lamb

 


 

 

 

Tuesday, September 10, 2013

 

Review and Updates of the USDA-APHIS Veterinary Services (VS) National Chronice Wasting Disease (CWD) Program 2012-2013

 


 

 

 
Sunday, August 11, 2013

 

Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013

 

Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010

 


 

 

 

Friday, August 16, 2013

 

*** Creutzfeldt-Jakob disease (CJD) biannual update August 2013 U.K. and Contaminated blood products induce a highly atypical prion disease devoid of PrPres in primates

 


 

 

 

Sunday, September 08, 2013

 

Iatrogenic Creutzfeldt-Jakob disease via surgical instruments and decontamination possibilities for the TSE prion

 


 

 

 

Thursday, September 26, 2013

 

Minimise transmission risk of CJD and vCJD in healthcare settings Guidance

 


 

 

 

pens, pens, PENS ??

 

*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.

 


 

 

now, decades later ;

 

 

2012

 

PO-039: A comparison of scrapie and chronic wasting disease in white-tailed deer

 

Justin Greenlee, Jodi Smith, Eric Nicholson US Dept. Agriculture; Agricultural Research Service, National Animal Disease Center; Ames, IA USA

 

snip...

 

The results of this study suggest that there are many similarities in the manifestation of CWD and scrapie in WTD after IC inoculation including early and widespread presence of PrPSc in lymphoid tissues, clinical signs of depression and weight loss progressing to wasting, and an incubation time of 21-23 months. Moreover, western blots (WB) done on brain material from the obex region have a molecular profile similar to CWD and distinct from tissues of the cerebrum or the scrapie inoculum. However, results of microscopic and IHC examination indicate that there are differences between the lesions expected in CWD and those that occur in deer with scrapie: amyloid plaques were not noted in any sections of brain examined from these deer and the pattern of immunoreactivity by IHC was diffuse rather than plaque-like. After a natural route of exposure, 100% of WTD were susceptible to scrapie. Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for PrPSc by IHC and WB. Similar to IC inoculated deer, samples from these deer exhibited two different molecular profiles: samples from obex resembled CWD whereas those from cerebrum were similar to the original scrapie inoculum. On further examination by WB using a panel of antibodies, the tissues from deer with scrapie exhibit properties differing from tissues either from sheep with scrapie or WTD with CWD. Samples from WTD with CWD or sheep with scrapie are strongly immunoreactive when probed with mAb P4, however, samples from WTD with scrapie are only weakly immunoreactive. In contrast, when probed with mAb’s 6H4 or SAF 84, samples from sheep with scrapie and WTD with CWD are weakly immunoreactive and samples from WTD with scrapie are strongly positive. This work demonstrates that WTD are highly susceptible to sheep scrapie, but on first passage, scrapie in WTD is differentiable from CWD.

 


 

 

2011

 

*** After a natural route of exposure, 100% of white-tailed deer were susceptible to scrapie.

 


 

 

Scrapie in Deer: Comparisons and Contrasts to Chronic Wasting Disease (CWD)

 

Justin J. Greenlee of the Virus and Prion Diseases Research Unit, National Animal Disease Center, ARS, USDA, Ames, IA

 

snip...

 

This highlights the facts that 1) prior to the onset of clinical signs PrPSc is widely distributed in the CNS and lymphoid tissues and 2) currently used diagnostic methods are sufficient to detect PrPSc prior to the onset of clinical signs. The results of this study suggest that there are many similarities in the manifestation of CWD and scrapie in white-tailed deer after IC inoculation including early and widespread presence of PrPSc in lymphoid tissues, clinical signs of depression and weight loss progressing to wasting, and an incubation time of 21-23 months. Moreover, western blots (WB) done on brain material from the obex region have a molecular profile consistent with CWD and distinct from tissues of the cerebrum or the scrapie inoculum. However, results of microscopic and IHC examination indicate that there are differences between the lesions expected in CWD and those that occur in deer with scrapie: amyloid plaques were not noted in any sections of brain examined from these deer and the pattern of immunoreactivity by IHC was diffuse rather than plaque-like. After a natural route of exposure, 100% of white-tailed deer were susceptible to scrapie. Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for scrapie by IHC and WB. Tissues with PrPSc immunoreactivity included brain, tonsil, retropharyngeal and mesenteric lymph nodes, hemal node, Peyer’s patches, and spleen. While two WB patterns have been detected in brain regions of deer inoculated by the natural route, unlike the IC inoculated deer, the pattern similar to the scrapie inoculum predominates.

 


 

 

2011 Annual Report

 

Research Project: TRANSMISSION, DIFFERENTIATION, AND PATHOBIOLOGY OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES Location: Virus and Prion Research Unit

 

2011 Annual Report

 

In Objective 1, Assess cross-species transmissibility of transmissible spongiform encephalopathies (TSEs) in livestock and wildlife, numerous experiments assessing the susceptibility of various TSEs in different host species were conducted. Most notable is deer inoculated with scrapie, which exhibits similarities to chronic wasting disease (CWD) in deer suggestive of sheep scrapie as an origin of CWD.

 

snip...

 

4.Accomplishments 1. Deer inoculated with domestic isolates of sheep scrapie. Scrapie-affected deer exhibit 2 different patterns of disease associated prion protein. In some regions of the brain the pattern is much like that observed for scrapie, while in others it is more like chronic wasting disease (CWD), the transmissible spongiform encephalopathy typically associated with deer. This work conducted by ARS scientists at the National Animal Disease Center, Ames, IA suggests that an interspecies transmission of sheep scrapie to deer may have been the origin of CWD. This is important for husbandry practices with both captive deer, elk and sheep for farmers and ranchers attempting to keep their herds and flocks free of CWD and scrapie.

 


 

 

White-tailed Deer are Susceptible to Scrapie by Natural Route of Infection

 

Jodi D. Smith, Justin J. Greenlee, and Robert A. Kunkle; Virus and Prion Research Unit, National Animal Disease Center, USDA-ARS

 

snip...

 

This work demonstrates for the first time that white-tailed deer are susceptible to sheep scrapie by potential natural routes of inoculation. In-depth analysis of tissues will be done to determine similarities between scrapie in deer after intracranial and oral/intranasal inoculation and chronic wasting disease resulting from similar routes of inoculation.

 

see full text ;

 


 

 

SEE MORE USAHA REPORTS HERE, 2012 NOT PUBLISHED YET...TSS

 


 


 


 

 

Thursday, June 20, 2013

 

atypical, BSE, CWD, Scrapie, Captive Farmed shooting pens (livestock), Wild Cervids, Rectal Mucosa Biopsy 2012 USAHA Proceedings, and CJD TSE prion Update

 


 

 

 

Tuesday, September 17, 2013

 

USAHA 116TH ANNUAL MEETING October 18 – 24, 2012 CWD, Scrapie, BSE, TSE prion (September 17, 2013)

 


 

 

 

*** The potential impact of prion diseases on human health was greatly magnified by the recognition that interspecies transfer of BSE to humans by beef ingestion resulted in vCJD. While changes in animal feed constituents and slaughter practices appear to have curtailed vCJD, there is concern that CWD of free-ranging deer and elk in the U.S. might also cross the species barrier. Thus, consuming venison could be a source of human prion disease. Whether BSE and CWD represent interspecies scrapie transfer or are newly arisen prion diseases is unknown. Therefore, the possibility of transmission of prion disease through other food animals cannot be ruled out. There is evidence that vCJD can be transmitted through blood transfusion. There is likely a pool of unknown size of asymptomatic individuals infected with vCJD, and there may be asymptomatic individuals infected with the CWD equivalent. These circumstances represent a potential threat to blood, blood products, and plasma supplies.

 

 


 

 

 

IT is of my opinion, that the OIE and the USDA et al, are the soul reason, and responsible parties, for Transmissible Spongiform Encephalopathy TSE prion diseases, including typical and atypical BSE, typical and atypical Scrapie, and all strains of CWD, and human TSE there from, spreading around the globe.

 

 

 

I have lost all confidence of this organization as a regulatory authority on animal disease, and consider it nothing more than a National Trading Brokerage for all strains of animal TSE, just to satisfy there commodity. AS i said before, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization.

 

 

 

JUST because of low documented human body count with nvCJD and the long incubation periods, the lack of sound science being replaced by political and corporate science in relations with the fact that science has now linked some sporadic CJD with atypical BSE and atypical scrapie, and the very real threat of CWD being zoonosis, I believed the O.I.E. has failed terribly and again, I call for this organization to be dissolved. ...

 

 

 
IN A NUT SHELL ;

 

(Adopted by the International Committee of the OIE on 23 May 2006)

 

11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,

 

 


 

 

 

Thursday, May 30, 2013

 

World Organization for Animal Health (OIE) has upgraded the United States' risk classification for mad cow disease to "negligible" from "controlled", and risk further exposing the globe to the TSE prion mad cow type disease

 

U.S. gets top mad-cow rating from international group and risk further exposing the globe to the TSE prion mad cow type disease

 


 


 

 

Tuesday, June 11, 2013

 

Weld County Bi-Products dba Fort Morgan Pet Foods 6/1/12 significant deviations from requirements in FDA regulations that are intended to reduce the risk of bovine spongiform encephalopathy (BSE) within the United States

 


 

 

 

Thursday, June 6, 2013

 

 

BSE TSE PRION USDA FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5, 2013

 

 

 

Greetings,

 

 

since our fine federal friends have decided not to give out any more reports on the USA breaches of the feed ban and surveillance etc. for the BSE TSE prion mad cow type disease in the USDA livestock, I thought I might attempt it. I swear, I just don’t understand the logic of the SSS policy, and that includes all of it. I assure you, it would be much easier, and probably better for the FDA and the USDA INC., if they would simply put some kind of report out for Pete’s sake, instead of me doing it after I get mad, because I am going to put it all out there. the truth.

 

 

PLEASE BE ADVISED, any breach of any of the above classifications OAI, VAI, RTS, CAN lead to breaches into the feed BSE TSE prion protocols, and CAN lead to the eventual suspect tainted feed reaching livestock. please, if any USDA official out there disputes this, please explain then how they could not. paperwork errors can eventually lead to breaches of the BSE TSE prion mad cow feed ban reaching livestock, or contamination and exposure there from, as well.

 

 

I would sure like to see the full reports of just these ;

 

 

 

4018 CHI-DO 3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR FR, OF HP 11/26/2012 OAI Y

 

 

9367 3008575486 Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO 81067 OPR RE, TH HP 2/27/2013 OAI N

 

 

9446 DEN-DO 1713202 Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO 80631-9501 OPR RE, TH HP 10/12/2012 OAI N

 

 

9447 DEN-DO 3002857110 Weld County Bi-Products dba Fort Morgan Pet Foods 13553 County Road 19 Fort Morgan CO 80701-7506 OPR RE HP 12/7/2011 OAI N

 

 

see full list of the fda mad cow bse feed follies, toward the bottom, after a short brief update on the mad cow bse follies, and our good friend Lester Crawford that was at the FDA.

 

 

ALSO, I would kindly like to comment on this FDA BSE/Ruminant Feed Inspections Firms Inventory (excel format)4 format, for reporting these breaches of BSE TSE prion protocols, from the extensive mad cow feed ban warning letters the fda use to put out for each violations. simply put, this excel format sucks, and the FDA et al intentionally made it this difficult to follow the usda fda mad cow follies. this is an intentional format to make it as difficult as possible to follow these breaches of the mad cow TSE prion safety feed protocols. to have absolutely no chronological or numerical order, and to format such violations in a way that they are almost impossible to find, says a lot about just how far the FDA and our fine federal friends will go through to hide these continued violations of the BSE TSE prion mad cow feed ban, and any breaches of protocols there from. once again, the wolf guarding the henhouse $$$

 

 

 

NAI = NO ACTION INDICATED

 

OAI = OFFICIAL ACTION INDICATED

 

VAI = VOLUNTARY ACTION INDICATED

 

RTS = REFERRED TO STATE

 

 

 

Inspections conducted by State and FDA investigators are classified to reflect the compliance status at the time of the inspection, based upon whether objectionable conditions were documented. Based on the conditions found, inspection results are recorded in one of three classifications:

 

OAI (Official Action Indicated) when inspectors find significant objectionable conditions or practices and believe that regulatory sanctions are warranted to address the establishment’s lack of compliance with the regulation. An example of an OAI classification would be findings of manufacturing procedures insufficient to ensure that ruminant feed is not contaminated with prohibited material. Inspectors will promptly re-inspect facilities classified OAI after regulatory sanctions have been applied to determine whether the corrective actions are adequate to address the objectionable conditions.

 

VAI (Voluntary Action Indicated) when inspectors find objectionable conditions or practices that do not meet the threshold of regulatory significance, but warrant an advisory to inform the establishment that inspectors found conditions or practices that should be voluntarily corrected. VAI violations are typically technical violations of the 1997 BSE Feed Rule. These violations include minor recordkeeping lapses or conditions involving non-ruminant feeds.

 

NAI (No Action Indicated) when inspectors find no objectionable conditions or practices or, if they find objectionable conditions, those conditions are of a minor nature and do not justify further actions.

 

 

 


 

 

 

when sound science was bought off by junk science, in regards to the BSE TSE prion mad cow type disease, by the USDA, CFIA, WHO, OIE, et al. $$$

 

when the infamous, and fraudulently USDA, FSIS, APHIS, FDA, gold card was taken away that infamous day in December of 2003, all cards were off the table, it was time to change the science, and change they did. ...tss

 

 

snip. ...please see full text ;

 

 

Thursday, June 6, 2013

 

 

BSE TSE PRION USDA FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5, 2013

 

 


 

 

 

Friday, July 19, 2013

 

PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED Revised as of April 1, 2013 50# Regular Chicken Feed was found to contain mammalian protein label does not contain the warning statement

 

 


 

 

 

 

Sent: Sunday, July 21, 2013 1:44 PM
Subject: Welsh Government and Food Standards Agency Wales Joint Public Consultation on the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013
 

July 21, 2013
 
 
 
Subject: Welsh Government and Food Standards Agency Wales Joint Public Consultation on the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013
 

endemics@wales.gsi.gov.uk ; fenris@caramail.com ;

 

 

Greetings Welsh Government and Food Standards Agency Wales,

 

 

With great urgency, I would kindly like to comment on ;

 
 

Number: WG18417

 
 
 
Joint Public Consultation on the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013

 

 
 
 
The European Food Safety Authority (EFSA) and the European Centre for Disease Prevention and Control jointly advised in 2011 that BSE is the only animal TSE that has been shown to be a risk to human health and that there is no epidemiological evidence to suggest that classical scrapie is a risk to human health.
 
 
 
and
 
 
 
 
What are the main issues under consideration?
 
2.1 The main issues under consideration relate to changes in BSE testing requirements; more proportionate measures for controlling classical scrapie in sheep flocks and goat herds in which classical scrapie is confirmed; and more proportionate controls on animal feed. The Welsh Government also wishes to consult on proposed amendments to the BSE cattle compensation system in light of identified anomalies in the current system, in addition to a variety of other proposed technical and procedural amendments to the 2008 Regulations.
 
2.2 The key specific amendments are summarised and then considered in more detail below. Other proposed technical amendments, which are considered to have a negligible impact or have already been implemented administratively, are listed at Annex A.

 

What are the main issues under consideration?
 
 
2.1
 
 
* The main issues under consideration relate to changes in BSE testing requirements;
 
 
* more proportionate measures for controlling classical scrapie in sheep flocks
 
 
* and goat herds in which classical scrapie is confirmed; and
 
 
* more proportionate controls on animal feed.
 
 
 
 
Greetings again Welsh Government and Food Standards Agency Wales,
 
 
 
with another TSE prion medical blunder happening just this past week ;
 


Friday, July 19, 2013

Beaumont Hospital in Dublin assessing patients for CJD http://creutzfeldt-jakob-disease.blogspot.com/2013/07/beaumont-hospital-in-dublin-assessing.html

 

 

I STRENUOUSLY urge you take my submission with the greatest urgency.

 

My submission and concerns as follows, and in part mixed in and throughout the  WG18417  Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013.

 

First and foremost, I think it is very important for the Welsh Government and Food Standards Agency Wales, to take a full inventory of your imports from North America, and other Countries, especially your pet and fish foods, for reasons and scientific facts I have supplied below.

 

I think the attempt by Governments around the globe to do away with the Transmissible Spongiform Encephalopathy TSE prion disease, via weakening of the BSE TSE prion surveillance, lowering of age limits in testing, weakening the feed bans, caving in to industry, ignoring all the science of the past 3 decades, ignoring these different atypical TSE prion disease breaking out, making up new names for these TSE prion disease, and by the way, what ever happened to the IBNC BSE, and the pathology there, and what about testing there from?

 

The USDA, CFIA, with the help of the OIE, have made it there goal to extinguish all BSE TSE prion trade barriers, before all the science on the TSE prion disease is in, and are working hard to exempt all TSE prion in all species from any trade barrier, and the OIE is working right along with them. This happened December 2003, when the USA lost it’s OIE BSE Gold Card, when BSE was first documented in the USA, after a long hard fought battle trying to cover mad cow disease up. This proven time and time again by the OIG and the GAO of the USA. I will supply url links of submissions I have made to the USDA et al over the years, since the death of my Mother to the Heidenhain Variant of Creutzfeldt Jakob Disease i.e. hvCJD ‘confirmed’ 12/14/97. just another happenstance of bad luck they tell me, that it only happens in the UK, and that it’s a UK disease, this mad cow disease, that no other animal species TSE prion disease in the world, at no other location, can transmit a TSE prion disease to a human, and then basing all trading protocol from country to country, based on this junk science, destroys the past 3 decades of trying to eradicate the damn disease, and the OIE, USDA, CFIA, and all the other countries that know better, that just goes along with this due to trade purposes, just because it’s a long incubating disease, just because the science is still in it’s infancy, does not mean we should start ignoring what early science has taught us, and start weakening any safety protocols there from.

 

North America has the most documented TSE prion disease in the wild and in farmed livestock than any other country in the world, excluding the TSE prion disease documented in zoo animals. Chronic Wasting Disease CWD in cervids is running rampant, the USA and Canada can’t stop it, while Mexico has not a clue of any TSE prion disease.  The shooting pens, and CWD there from in the USA, is a real risk factor, one the game farms refuse to admit they are a big problem with the spreading of the CWD TSE prion disease, fighting tooth and nail completely ignoring the evolving science on the CWD TSE prion disease, and how it’s spread, and these antler mills are multiplying from state to state in big numbers. There is ample evidence of CWD transmission to humans, to warrant a warning to the world, of the IATROGENIC potential for this TSE prion disease in cervids, via the multitude of potential routes of infection, via the medical, dental, surgical, blood, tissue.  CWD has now mutated to multiple strains. The science is there to warrant this very real concern, it’s just the same as with what happened in the U.K., the industry and USDA inc., are stopping these concerns to be made public, with the same watered down junk science used in the beginning of the BSE blunder.  you should all be very aware of this, if you come abroad to North America. DEFRA has put out a warning on CWD TSE prion disease in the USA and have put out a document I supplied with additional risk factors from North America, this is supplied below as well.

 

Another concern is with your assumptions that typical classical scrapie is not a risk factor for humans, when there _is_ evidence to show otherwise, that indeed typical scrapie is a risk factor for humans, as with atypical Scrapie.

 

The OIE, USDA inc, and the CFIA, have come to the conclusion that neither typical scrapie nor the atypical Nor-98 scrapie are neither a risk factor for humans, they have urged the OIE to conclude that atypical Nor-98 to be exempt from any trading protocols, and indeed have made the Nor-98 atypical scrapie EXEMPT, and made it legal to trade, and they are also in the works to make typical scrapie exempt.

 

typical scrapie consist of many different strains of scrapie, not just one. and the atypical Nor-98 has very similar features with human Gerstmann-Sträussler-Scheinker Disease GSS and Variably Protease-Sensitive Prionopathy VPSPr. I have not seen anywhere in the Bible, or the scrolls of the Dead Sea, where it was stipulated that indeed typical c-BSE is the only zoonosis Transmissible Spongiform Encephalopathy TSE prion disease. This is ludicrous in 2013 to still believe this junk science.

 

With the science to date of the 1st 10 nvCJD victims of Dr. Ironside et al, and the diagnostic criteria then to diagnose the nvCJD, compared to what Dr. Gambetti et al diagnosed in their 1st 10, of which young victims are being diagnosed, but yet changed the name to VPSPr type CJD human TSE, is not scientific in my opinion. I believe that the UKBSEnvCJD only theory is bogus, it is not scientific, and should be put to bed once and for all. you cannot have your cake and eat it too. either Ironside was wrong, or Gambetti is wrong. to continue this UKBSEnvCJD only myth, will only help continue spread the TSE prion agent long and far.

 

typical Scrapie has been studied for decades and decades, and has proven to be transmitted to non-human primates by their NON-FORCED oral consumption (Gibbs et al).  when you write in absolute terms as this is fact that  ‘’BSE is the only animal TSE that has been shown to be a risk to human health and that there is no epidemiological evidence to suggest that classical scrapie is a risk to human health’’ may be true in terms of documentation, but in terms of science to date, I think you are wishing. I kindly wish to submit the following in good faith ;

 

 

snip...

 

 

Singeltary Complete submission located in PDF attachment in this email, or in the link below...

 
 
 
 
 
Sunday, July 21, 2013
 
Welsh Government and Food Standards Agency Wales Joint Public Consultation on the Proposed Transmissible Spongiform Encephalopathies (Wales) Regulations 2013 Singeltary Submission WG18417
 
 
 
 
TSS
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