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Live Blood Analysis

Posted Aug 26 2009 11:08pm
Found out my NET practitioner in Newport Beach has also been a Darkfield Microscopist since 1996, so decided to spend half of my appt with her analyzing my blood. Here is my post from my previous live blood analysis in Germany

I wish I'd gotten pictures of my analysis this time, because I just realized now how different my blood looks. On one hand, I have far less pathogens in my blood (she didn't find any mycoplasma--she was shocked--, viruses, and only a few fungal forms), but I had Rouleau, or erythrocyte aggregation going on. This is more commonly known in the CFIDS community as hypercoagulation. She gave me 6 capsules of 90,000units Protease each to take, took my blood again 10 minutes later, and all the couch-potato red blood cells started to move not surprisingly. (Hemocare is the brand, and each capsule has 34,000 more units of Protease than 3 tablets of Wobenzyme--impressive product)

I have a few target cells (they have a big white chunk in the middle) which supposedly means I'm not uptaking minerals. This is not surprising considering the ion channels of our cells barely work.

Some bowel toxins (GI issues), uric acid crystals (kidney's inefficient at breaking down purines), more monocytes than neutrophils (immune inflammation->another sign of immune dysfunction). There were a few protoplasts--strings of residue emitted by bacteria. More on this later. Liver looks good.

So my two cents: the only pathogenic forms were protoplasts and some fungal forms. Protoplasts: "The living material of a plant or bacterial cell, including the protoplasm and plasma membrane after the cell wall has been removed." Either bacteria are mutating or on the defensive. My hunch is that they're on their last line of defense since I really haven't used any antimicrobials at all lately besides electromagnetic frequency machines, which are far less likely to induce resistance & hence mutation. So this just adds to the growing body of evidence I have that I have infections pretty well under control and my main focus now is and should be on cellular metabolism.

RBCs clumped together: anaerobic metabolism and in laymen's terms: my cells are acting like I just told them to bust rocks on a chain gang. I think the high-dose protease enzymes will help get things moving, but I'm not shortsighted enough to consider that a permanent solution. Peterson said we have "acquired mitochondrial dysfunction," and he does not mean "acquired" in the HIV sense. ANS and immune dysfunction--don't fail me now.
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