Psychiatric Medications and Immunity: IgG and IgE

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Psychiatric Medication Failures: Stealth Messages From Your IgG
The longer we deeply explore treatment disappointments [that occur everyday], the more we see significant confusion and missed diagnosis with psych med failures secondary to overlooking IgG, the “chronic antibody.”

IgE is the most frequently tested antibody – sought out when the allergists are looking for an acute response [think: E = Emergency] to an invader trying to get through your barriers [e.g. skin, lungs, bowels]. IgG, on the other hand, is the most overlooked messenger, the night-time carrier pigeon that reveals underlying allergens at play in the unsuspecting victim through years of very few or no physical symptoms secondary to stealth allergens. IgE can herald obvious medication reactions, IgG messengers aren’t so obvious.

IgG antibody reactions to stealth invaders most often fly below your searching affect-sensory, pain-sensory, and thinking-sensory radar. If IgG is at play backstage, reacting to antigens that are corrupting your system [like casein and gluten ], including your s and your hormones, both feeling and thinking can become significantly unbalanced. Cytokines have a field day and over time the feeling-good party is over.

Symptom Recognition

If you can’t see or feel it it must not be a problem? I don’t think so. Quite conspicuously, the bowel has few nerve endings, as often seen in Bowel CA presentations, – so one can have a significant problem and for years remain unaware of the downstream effects.

From Great Plains Laboratory comes this explanation:

Example of a Typical IgE Response:
– Suppose a person with a peanut allergy eats a peanut. B cells in the body are exposed to the peanut allergens. B cells begin making IgE antibodies to fight the peanut “infection”. These IgE antibodies were made specifically for defending the body against peanuts. The IgE antibodies bind to the peanut molecules or allergens in the body.

After the exposure to peanuts, IgE antibodies can also attach themselves to mast cells. There the IgE antibodies wait until the next peanut exposure. When the next peanut exposure occurs the IgE antibodies signal the mast cells to release histamine and other compounds. Histamine and these other compounds are the cause of allergy symptoms like itching and inflammation. All of this usually happens within minutes of ingesting the allergen. IgE allergies are treated with medications that block the release of histamines.

What are the main causes of IgG or delayed food allergies?

Digestive problems play a major role in the development of IgG food allergies. Some individuals have leaky guts, meaning that food particles are able to enter the bloodstream. When these particles are recognized by the immune system the body has an immune response. More antibodies are created to fight the perceived infection of the foreign food particles. This can lead to a weakening of the overworked immune system.

IgG food allergies are treated by removing problem foods from the diet and by helping digestion with probiotics.

In quick summary these issues, my friends, are a significant problem, see everyday in our offices – and if not understood, then not considered or tested, the corruption can go on for a lifetime.

If you are already thinking about these matters take your thoughts to this next step regarding returning to eat that offending antigen:

Interesting notes, more exploration will prove useful,
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